Sunday Studies - day 173 2/3 :)

It's just been physiology today with a nice break in between to have breakfast with my parents.

forgot to post my attendance grade from last week ...

Week 9 Attendance = 97%

3 workouts = 100%

of course, i can get all three workouts in under an hour total time ....

I wanted to go over the disorders pertaining to many of the hormones we're studying in physio, I haven't done that yet and thought it might make for an interesting post. Basically, a disorder exist when we have too much (hyper) of a hormone or too little (hypo) of a hormone and sometimes, the disorder depends on how old we are when we have too much or too little - Let's see what we've got!

Diabetes Insipidus - Lack of antidiuretic hormone ADH (vasopresson) from the posterior pituitary

Hormones from the Anterior Pituitary

Grown Hormone Disorders -

1. Acromegaly - too much growh hormone GH after puberty
2. Gigantism - too much grown hormone GH before puberty
3. Pituitary Dwarfism - decreased levels of GH before puberty
4. Diabetes Mellitus - because of increased glucose levels we see diabetogenic effects that can become diabetes if insulin activity can't occur.

Adrenocorticotropic Hormone (ACTH)

1. Addison's Disease - decreased levels of corticohormones
2. Cushing's Disease - increased levels of corticohormones

ACTH stimulates cells of the adrenal cortex that produce clucocorticoids, especially cortisol which is how ACTH is ultimately responsible for those aforementioned diseases.

Thyroid-Stimulating Hormone (TSH)

1. Myxedema - decreased levels of TSH in adults (hypothyroidism)
2. Cretinism - decreased levels of TSH in children (hypothyroidism)
3. Hyperthyroidism - increased levels of TSH

Melatonin (from Pineal Gland)

1. Seasonal Affective Disorder (SAD)
2. Jet Lag

Thyroid hormones (T3 & T4)

1. Hypothyroidism
2. Hyperthyroidism
3. Graves Disease, autoimmune hyperthyroidism
4. Goiter (from either hyper or hypothyroidism)

Parathyroid Gland

1. Hyperparathyroidism

Adrenal Glands

1. hyperplasia
2. Addison's & Cushings dz would also be listed here but the impetus for increase or decrease of corticohormones is from the ACTH previously mentioned

Pancreatic Islets

1. Beta cells, Insulin - Diabetes Mellitus
2. Type I - defective beta cells, hence, decrease in insulin productive, could be due to autoimmune disorder or viral infection
3. Type II - decreased sensitivity to insulin

I guess that's good enough for now, it's almost 2 a.m. and I am WAY past my bedtime.