Deleting a few Pages - NMS

Over along the right hand column have been a few pages that don't exactly fit anymore so I'll be deleting a few of them, specifically the pages dealing with Tri-5 and classes such as Physical Diagnosis I, Toxicology (pharmacology), Biomechanics and NMS.  However, in case any of these pages could be of use to somebody I'm going to save each page as a post.

Below are the contents from the deleted Tri-5 NMS Page:

• agnosia - loss of discriminatory ability in an extremity
• akinesthesia - inability to discriminate the position of a finger or toe in space
• Babinski sign - classic example of a pathological reflex which means the corticospinal tract is lesioned. (p13)
• dural test - WLR, Soto-Hall, Valsalva
• hyperreflexia - often seen with umnl due to lack of inhibition from the CNS on the alpha motor neuron
• kinesthesia - the ability to know where a digit (finger/toe) is in space.  With eyes closed the only way this type information could get to the cortex would be over the lemniscal pathway.
• lemniscal lesion - eliminate proprioception but spare nociception (p4)
• parasthesia - tingling sensation
• radiculopathy - a lesion of the nerve root; when the nerve root is being compressed by a particular entity (spur, ligament, disc, narrowed IVF, etc.)
• spastic paralysis - is due to loss of pyramidal (corticospinal) pathway
• spasticity - due to loss of extrapyramidal inhibitory influences
• thalamus lesion - loss of both pain & proprioception
• Tinel Sign - pain & tingling radiating distally upon irritation of a lesion site (p7)

Signs of an Upper Motor Neuron Lesion (UMNL)

 - paralysis

An UMNL will manifest as damage to both the pyramidal and extrapyramidal tracts.

Lower Motor Neuron Lesion (LMNL) - 

flaccid paralysis (flaccidity = loss of reflexes)

The effect of a lower motor neuron lesion on muscle tone is due to decreased excitation

Bell's Palsy is one such dz which is due to a LMNL